2016 Reviewer and Editorial Board Thank You
article has not abstract
Published in the journal:
. PLoS Genet 13(3): e32767. doi:10.1371/journal.pgen.1006671
Category:
Editorial
doi:
https://doi.org/10.1371/journal.pgen.1006671
Summary
article has not abstract
PLOS and the PLOS Genetics team would like to express our sincere gratitude to everyone who lent us their expertise as an associate editor, as a guest editor, or as a peer reviewer in 2016. We are fully dependent on the voluntary efforts of these experts to publish the journal for the academic community. Over the past year, PLOS Genetics relied on 654 associate and guest editors to manage the 2,474 articles submitted to the journal, which were assessed by nearly 3,000 reviewers. Their efforts enabled the publication of more than 680 rigorous, Open Access papers.
The names of our 2016 editors that handled submitted manuscripts appear in the Supporting Information as S1 Editor List and as S1 Guest Editor List. Our reviewers appear in the Supporting Information as S1 Reviewer List. We are grateful for their dedication, generosity, and support of Open Access science. Thank you all.
Supporting Information
Zdroje
1. (2016) PLOS Genetics 2015 Reviewer Thank You. PLoS Genet 12(2): e1005925.
2. (2015) PLOS Genetics 2014 Reviewer Thank You. PLoS Genet 11(2): e1005070.
Štítky
Genetika Reprodukční medicínaČlánek vyšel v časopise
PLOS Genetics
2017 Číslo 3
- Primární hyperoxalurie – aktuální možnosti diagnostiky a léčby
- Srdeční frekvence embrya může být faktorem užitečným v předpovídání výsledku IVF
- Akutní intermitentní porfyrie
- Vztah užívání alkoholu a mužské fertility
- Šanci na úspěšný průběh těhotenství snižují nevhodné hladiny progesteronu vznikající při umělém oplodnění
Nejčtenější v tomto čísle
- CUZD1 is a critical mediator of the JAK/STAT5 signaling pathway that controls mammary gland development during pregnancy
- A rare loss-of-function mutation reduces blood eosinophil counts and protects from asthma
- A variant in the gene in a dog with ichthyosis
- Fishing for adaptive epistasis using mitonuclear interactions
