SEVERE NEAR REFLEX SPASM IN A HEALTHY TEENAGER. A CASE REPORT

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Autoři: G. Mejía ^1,2; S. Guevara ²; Y. Salgado ²; A. Tello ^3,4,5
Působiště autorů: Neuroscience Research Group (NEUROS). Neurovitae Research Center. School of Medicine and Health Sciences. Rosario University, Bogotá, Colombia ¹; Ophthalmology Interest Group Universidad del Rosario. (OIG UR). School of Medicine and Health Sciences. Rosario University, Bogotá, Colombia ²; Virgilio Galvis Eye Center, Floridablanca, Colombia ³; Ophthalmology Foundation of Santander - Carlos Ardila Lulle (FOSCAL), Floridablanca, Colombia ⁴; Ophthalmology Foundation of Santander, Industrial University of Santander (UIS), Bucaramanga, Colombia ⁵
Vyšlo v časopise: Čes. a slov. Oftal., 79, 2023, No. 2, p. 97-100
Kategorie: Kazuistika
doi: https://doi.org/10.31348/2023/15

Souhrn

Introduction: Spasm of the near reflex usually includes accommodative spasm, esophoria/tropia, and different degrees of miosis. Patients usually refer to distance blurred and fluctuating vision, ocular discomfort, and headaches. The diagnosis is established with refraction with and without cycloplegia; most of the cases have a functional etiology. However, some cases require neurological conditions to be ruled out; cycloplegics have an important diagnostic and therapeutic role.

Purpose: To describe a case of bilateral severe accommodative spasm in a healthy 14-year-old teenager.

Case presentation: A 14-year-old boy with progressive diminished visual acuity attended for YSP consultation. The diagnosis of bilateral spasm of the near reflex was made, based on a gap refraction of 9.75 D between retinoscopy with and without cycloplegia and esophoria with normal keratometry and axial length. The spasm was eliminated with 2 drops of cycloplegic in each eye separated by 15 days; no clear etiology was found other than the start of school.

Conclusion: Clinicians should be aware of pseudomyopia, especially in children with acute changes in visual acuity, who are usually exposed to myopigenic environmental factors that induce overstimulation of the parasympathetic third cranial nerve’s innervation.

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