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ReklamaLack of reproducibility in osteocalcin-deficient mice
Autoři: Takeshi Moriishi aff001; Toshihisa Komori aff002
Působiště autorů: Department of Cell Biology, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki, Japan aff001; Basic and Translational Research Center for Hard Tissue Disease, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki, Japan aff002
Vyšlo v časopise: Lack of reproducibility in osteocalcin-deficient mice. PLoS Genet 16(6): e32767. doi:10.1371/journal.pgen.1008939
Kategorie: Formal Comment
doi: https://doi.org/10.1371/journal.pgen.1008939
Zdroje
1. Ducy P, Desbois C, Boyce B, Pinero G, Story B, Dunstan C, et al. Increased bone formation in osteocalcin-deficient mice. Nature. 1996;382(6590):448–52. Epub 1996/08/01. doi: 10.1038/382448a0 8684484.
2. Moriishi T, Ozasa R, Ishimoto T, Nakano T, Hasegawa T, Miyazaki T, et al. Osteocalcin is necessary for the alignment of apatite crystallites, but not glucose metabolism, testosterone synthesis, or muscle mass. PLoS Genet. 2020;16(5):e1008586. Epub 2020/05/29. doi: 10.1371/journal.pgen.1008586 32463816.
3. Diegel CR, Hann S, Ayturk UM, Hu JCW, Lim KE, Droscha CJ, et al. An osteocalcin-deficient mouse strain without endocrine abnormalities. PLoS Genet. 2020;16(5):e1008361. Epub 2020/05/29. doi: 10.1371/journal.pgen.1008361 32463812.
4. Bailey S, Karsenty G, Gundberg C, Vashishth D. Osteocalcin and osteopontin influence bone morphology and mechanical properties. Ann N Y Acad Sci. 2017;1409(1):79–84. Epub 2017/10/19. doi: 10.1111/nyas.13470 29044594; PubMed Central PMCID: PMC5730490.
5. Ducy P, Starbuck M, Priemel M, Shen J, Pinero G, Geoffroy V, et al. A Cbfa1-dependent genetic pathway controls bone formation beyond embryonic development. Genes Dev. 1999;13(8):1025–36. Epub 1999/04/24. doi: 10.1101/gad.13.8.1025 10215629; PubMed Central PMCID: PMC316641.
6. Maruyama Z, Yoshida CA, Furuichi T, Amizuka N, Ito M, Fukuyama R, et al. Runx2 determines bone maturity and turnover rate in postnatal bone development and is involved in bone loss in estrogen deficiency. Dev Dyn. 2007;236(7):1876–90. Epub 2007/05/15. doi: 10.1002/dvdy.21187 17497678.
7. Vella A, Kumar R. Osteocalcin and the Regulation of Glucose Metabolism. Clin Rev Bone Miner Metab. 2013;11(1):11–6. Epub 2013/03/01. doi: 10.1007/s12018-012-9126-x 26109922; PubMed Central PMCID: PMC4476242.
8. Gundberg CM, Lian JB, Booth SL. Vitamin K-dependent carboxylation of osteocalcin: friend or foe? Adv Nutr. 2012;3(2):149–57. Epub 2012/04/21. doi: 10.3945/an.112.001834 22516722; PubMed Central PMCID: PMC3648715 interest.
9. Tura A, Pacini G, Yamada Y, Seino Y, Ahrén B. Glucagon and insulin secretion, insulin clearance, and fasting glucose in GIP receptor and GLP-1 receptor knockout mice. Am J Physiol Regul Integr Comp Physiol. 2019;316(1):R27–r37. Epub 2018/11/22. doi: 10.1152/ajpregu.00288.2018 30462524.
10. Nicholson A, Reifsnyder PC, Malcolm RD, Lucas CA, MacGregor GR, Zhang W, et al. Diet-induced obesity in two C57BL/6 substrains with intact or mutant nicotinamide nucleotide transhydrogenase (Nnt) gene. Obesity (Silver Spring, Md). 2010;18(10):1902–5. Epub 2010/01/09. doi: 10.1038/oby.2009.477 20057372; PubMed Central PMCID: PMC2888716.
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